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by admin on June 3, 2010

 

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Mild Cognitive Impairment (MCI)–Telltale Signs That You May Be At Increased Risk for Dementia or Alzheimer’s Disease

by admin on July 19, 2009

By Ralph Sanchez, L.Ac.,CNS,D.Hom

Long before a diagnosis of Dementia or Alzheimers disease (AD) is given, brain damage is occurring that leads to, and reflects the gradual downward spiral of mental decline that typifies AD. A cluster of signs that define Mild Cognitive Impairment (MCI), especially memory loss and forgetfulness, may be the early warning indicators that you are at risk for developing AD. If you can visualize a spectrum whereby healthy brain/cognitive function in an individual is on one side of the spectrum, and AD is on the opposite side, MCI would be inching ever so close to the AD side of the spectrum. The individual with MCI is more functional than one who is clinically diagnosed with dementia, albeit with MCI, there is subtle to noticeable problems with memory, judgment, language and other cognitive skills. These symptoms, along with other criteria that define MCI, are now recognized as the transitional stage of brain degeneration that may lead to Dementia and AD. (1) [click to continue…]

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Blueberry Polyphenols Protect the Brain from the Degenerative Processes Associated with Brain Aging and Alzheimer’s Disease

February 3, 2011

By Ralph Sanchez, L.Ac.,CNS,D.Hom
The study of plant and fruit polyphenols, a rich source of dietary antioxidants, represents one of the most promising areas of research in the field of anti-aging, and the prevention of degenerative disorders such as Alzheimer’s disease. Recent and ongoing research indicates that polyphenols present in berries and other fruits and vegetables [...]

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ApoE4 Gene Link to Alzheimer’s Disease

July 19, 2010
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Brain-Derived Neurotrophic Factor – Growth Factor Demonstrates Promise in the Protection Against Alzheimer’s Disease Progression

October 17, 2009

By Ralph Sanchez, L.Ac.CNS,D.Hom.
One of the longest held thoughts about damage to our brain’s cells (neurons) was that once they were lost, it was a fait accompli. Damaged neurons were considered damaged goods, never to regain their function, and without any hope for regeneration. Fortunately, for those concerned about recovery from brain trauma, stroke, and [...]

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Alpha lipoic acid Protects Brain Cells–Antioxidant Mechanisms For Alzheimer’s Prevention

January 11, 2009

By Ralph Sanchez, L.Ac.,CNS,D.Hom.
Alpha lipoic acid (ALA), a naturally occurring nutrient found in many foods and available in supplemental form, is also synthesized in humans where it serves in energy metabolism and as a vital antioxidant. ALA is a unique antioxidant in that it is both water and fat-soluble, which enables ALA to confer its [...]

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Insulin, Brain Function And Alzheimer’s Disease – Is Insulin Resistance To Blame For Alzheimer’s?

September 21, 2008

By Ralph Sanchez, L.Ac.,CNS,D.Hom
Insulin fulfills an indispensable role in your body’s utilization of blood sugar (glucose). In type 2 diabetes and Metabolic Syndrome, insulin’s function of glucose uptake into the body’s cells is impaired due to a resistance to insulin that develops over time. This insulin resistance pattern which defines the disease process of the [...]

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Podcast Test

August 1, 2008

I will be starting a podcast on a regular basis.
This is a test to see how the system we’ve implemented is working.

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The Alzheimer’s Gene Puzzle – Genetic Links To Late Onset Alzheimer’s Disease (Part 1)

July 17, 2008

By Ralph Sanchez, L.Ac.,CNS,D.Hom
Genetic risk factors to Late Onset Alzheimer’s Disease (LOAD) are significant. A recent study of nearly 12,000 Swedish twin pairs, age 65 and older, determined that 58% to 79% of Alzheimer’s risk is genetic (1). This study showed that in male identical twins, when one brother had Alzheimer’s disease, the other developed [...]

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Plaques and Tangles in the Alzheimer’s Brain-Which One Is Most To Blame For Alzheimer’s Disease?

June 8, 2008

By Ralph Sanchez, L.Ac., CNS, D.Hom.
The two hallmark lesions that are associated with the damage that occurs in Alzheimer’s disease (AD) are neurofibrillary tangles and amyloid plaques (see pic 1). Processes involving inflammation, oxidative stress, * mitochondrial dysfunction, ** brain cholesterol dynamics (1) and others are tied into the formation of plaques and tangles. However, [...]

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